total erk cell signaling technology Search Results


total erk  (Cell Signaling Technology Inc)
99
Cell Signaling Technology Inc total erk
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Total Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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total erk (αerk  (Cell Signaling Technology Inc)
90
Cell Signaling Technology Inc total erk (αerk
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Total Erk (αerk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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map kinase erk1  (Cell Signaling Technology Inc)
96
Cell Signaling Technology Inc map kinase erk1
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Map Kinase Erk1, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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erk antibody  (Cell Signaling Technology Inc)
97
Cell Signaling Technology Inc erk antibody
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Erk Antibody, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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antibodies specific for dual phosphorylated-erk (thr204, tyr204) and total erk  (Cell Signaling Technology Inc)
90
Cell Signaling Technology Inc antibodies specific for dual phosphorylated-erk (thr204, tyr204) and total erk
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Antibodies Specific For Dual Phosphorylated Erk (Thr204, Tyr204) And Total Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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total erk rabbit polyclonal antibodies  (Cell Signaling Technology Inc)
97
Cell Signaling Technology Inc total erk rabbit polyclonal antibodies
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Total Erk Rabbit Polyclonal Antibodies, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 97/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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total erk  (Santa Cruz Biotechnology)
90
Santa Cruz Biotechnology total erk
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Total Erk, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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total 44 42 erk  (Cell Signaling Technology Inc)
93
Cell Signaling Technology Inc total 44 42 erk
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Total 44 42 Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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rabbit anti p44 p42 total erk  (Cell Signaling Technology Inc)
95
Cell Signaling Technology Inc rabbit anti p44 p42 total erk
Fig. 2. Effect of CCL2 treatment on <t>ERK,</t> <t>p38</t> and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).
Rabbit Anti P44 P42 Total Erk, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


Fig. 2. Effect of CCL2 treatment on ERK, p38 and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).

Journal: Osteoarthritis and Cartilage Open

Article Title: CCL2 induces articular chondrocyte MMP expression through ERK and p38 signaling pathways

doi: 10.1016/j.ocarto.2020.100136

Figure Lengend Snippet: Fig. 2. Effect of CCL2 treatment on ERK, p38 and JNK phosphorylation in normal chondrocytes. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 5, 10 or 15 min. FN-f (1 μM) treatment for 30 min was used as positive control. (A) Cell lysates were subjected to immu- noblotting to detect phospho-ERK, phospho-p38 and phospho-JNK. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK, p38 and JNK normalized to their respective loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ns ¼ not significant).

Article Snippet: Antibodies to phospho-ERK (Thr202/Tyr204) (#9101), total-ERK (#9102), phospho-p38 (Thr180/Tyr182) (#9211), total-p38 (#9212), phospho-JNK (Thr183/Tyr185) (#9253), total JNK (#9252) and MMP2 (#4022) were purchased from Cell Signaling Technology.

Techniques: Phospho-proteomics, Recombinant, Positive Control, Western Blot

Fig. 3. Effect of CCL2 treatment on ERK and p38 phosphorylation in normal chondrocyte with or without CCR2 inhibition. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 10 min. Where indicated, chondrocytes were pre-incubated with RS504393 (20 μM) for 1h prior to CCL2 treatment. (A) Immunoblotting analyses were performed to detect phospho-ERK and phospho-p38. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK and p38 with or without CCR2 inhibition normalized to their respecting loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots).

Journal: Osteoarthritis and Cartilage Open

Article Title: CCL2 induces articular chondrocyte MMP expression through ERK and p38 signaling pathways

doi: 10.1016/j.ocarto.2020.100136

Figure Lengend Snippet: Fig. 3. Effect of CCL2 treatment on ERK and p38 phosphorylation in normal chondrocyte with or without CCR2 inhibition. Confluent normal human articular chondrocytes were treated with recombinant CCL2 (20ng/ml) for 10 min. Where indicated, chondrocytes were pre-incubated with RS504393 (20 μM) for 1h prior to CCL2 treatment. (A) Immunoblotting analyses were performed to detect phospho-ERK and phospho-p38. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK and p38 with or without CCR2 inhibition normalized to their respecting loading controls. Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots).

Article Snippet: Antibodies to phospho-ERK (Thr202/Tyr204) (#9101), total-ERK (#9102), phospho-p38 (Thr180/Tyr182) (#9211), total-p38 (#9212), phospho-JNK (Thr183/Tyr185) (#9253), total JNK (#9252) and MMP2 (#4022) were purchased from Cell Signaling Technology.

Techniques: Phospho-proteomics, Inhibition, Recombinant, Incubation, Western Blot

Fig. 6. Effect of CCL2 treatment on expression of MMPs in OA chondrocytes in presence or absence of ERK and p38 inhibition. Human OA chondrocytes were treated with recombinant CCL2 (20ng/ml) for 10 min. Where indicated, chondrocytes were pre-incubated with ERK inhibitor (U0126, 10 μM) or p38 inhibitor (SB203580, 10 μM), for 1h prior CCL2 treatment. (A) Cell lysates were subjected to immunoblotting to detect phospho-ERK and phospho-p38. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK and p38 with or without ERK or p38 inhibition normalized to their respective loading controls. (C) Quantitative RT-PCR analyses were performed using the following probes: Mmp3 (n ¼ 6) and Mmp13 (n¼6). Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ####p 0.0001).

Journal: Osteoarthritis and Cartilage Open

Article Title: CCL2 induces articular chondrocyte MMP expression through ERK and p38 signaling pathways

doi: 10.1016/j.ocarto.2020.100136

Figure Lengend Snippet: Fig. 6. Effect of CCL2 treatment on expression of MMPs in OA chondrocytes in presence or absence of ERK and p38 inhibition. Human OA chondrocytes were treated with recombinant CCL2 (20ng/ml) for 10 min. Where indicated, chondrocytes were pre-incubated with ERK inhibitor (U0126, 10 μM) or p38 inhibitor (SB203580, 10 μM), for 1h prior CCL2 treatment. (A) Cell lysates were subjected to immunoblotting to detect phospho-ERK and phospho-p38. Immunoblots are representative of n¼4 independent donors. (B) Results of the densitometric analysis showing the effect of CCL2 treatment on the phosphorylation of ERK and p38 with or without ERK or p38 inhibition normalized to their respective loading controls. (C) Quantitative RT-PCR analyses were performed using the following probes: Mmp3 (n ¼ 6) and Mmp13 (n¼6). Data are presented as mean values SD. Statistical significance was set at p 0.05 (Asterisks represent 1way ANOVA summary; p values obtained with Tukey’s multiple comparisons are indicated on plots; ####p 0.0001).

Article Snippet: Antibodies to phospho-ERK (Thr202/Tyr204) (#9101), total-ERK (#9102), phospho-p38 (Thr180/Tyr182) (#9211), total-p38 (#9212), phospho-JNK (Thr183/Tyr185) (#9253), total JNK (#9252) and MMP2 (#4022) were purchased from Cell Signaling Technology.

Techniques: Expressing, Inhibition, Recombinant, Incubation, Western Blot, Phospho-proteomics, Quantitative RT-PCR